Autophagy facilitates an IFN-γ response and signal transduction

Yu Ping Chang; Chia Ling Chen; Su O. Chen; Yee Shin Lin; Cheng Chieh Tsai; Wei Ching Huang; Chi Yun Wang; Chia Yuan Hsieh; Pui Ching Choi; Chiou Feng Lin

doi:10.1016/j.micinf.2011.05.008

Autophagy facilitates an IFN-γ response and signal transduction

Yu Ping Chang, Chia Ling Chen, Su O. Chen, Yee Shin Lin, Cheng Chieh Tsai, Wei Ching Huang, Chi Yun Wang, Chia Yuan Hsieh, Pui Ching Choi, Chiou Feng Lin

微生物學科暨微生物及免疫學研究所

研究成果: Short survey › 同行評審

13 引文斯高帕斯（Scopus）

摘要

Autophagy, that is directly triggered by invaded pathogens and indirectly triggered by IFN-γ, acts as a defense by mediating intracellular microbial recognition and clearance. In addition, autophagy contributes to inflammation by facilitating an IFN-γ response and signal transduction. For immune escape, downregulated autophagy may be a strategy used by microbes.

原文	English
頁（從 - 到）	888-894
頁數	7
期刊	Microbes and Infection
卷	13
發行號	11
DOIs	https://doi.org/10.1016/j.micinf.2011.05.008
出版狀態	Published - 2011 10月

All Science Journal Classification (ASJC) codes

微生物學
免疫學
傳染性疾病

UN SDG

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10.1016/j.micinf.2011.05.008

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@article{6531d01a059b4b12a5c69dfee6d3aad7,

title = "Autophagy facilitates an IFN-γ response and signal transduction",

abstract = "Autophagy, that is directly triggered by invaded pathogens and indirectly triggered by IFN-γ, acts as a defense by mediating intracellular microbial recognition and clearance. In addition, autophagy contributes to inflammation by facilitating an IFN-γ response and signal transduction. For immune escape, downregulated autophagy may be a strategy used by microbes.",

author = "Chang, {Yu Ping} and Chen, {Chia Ling} and Chen, {Su O.} and Lin, {Yee Shin} and Tsai, {Cheng Chieh} and Huang, {Wei Ching} and Wang, {Chi Yun} and Hsieh, {Chia Yuan} and Choi, {Pui Ching} and Lin, {Chiou Feng}",

note = "Funding Information: This work was supported by the grant NHRI-EX99-9917NC from the National Health Research Institutes in Taiwan.",

year = "2011",

month = oct,

doi = "10.1016/j.micinf.2011.05.008",

language = "English",

volume = "13",

pages = "888--894",

journal = "Microbes and Infection",

issn = "1286-4579",

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TY - JOUR

T1 - Autophagy facilitates an IFN-γ response and signal transduction

AU - Chang, Yu Ping

AU - Chen, Chia Ling

AU - Chen, Su O.

AU - Lin, Yee Shin

AU - Tsai, Cheng Chieh

AU - Huang, Wei Ching

AU - Wang, Chi Yun

AU - Hsieh, Chia Yuan

AU - Choi, Pui Ching

AU - Lin, Chiou Feng

N1 - Funding Information: This work was supported by the grant NHRI-EX99-9917NC from the National Health Research Institutes in Taiwan.

PY - 2011/10

Y1 - 2011/10

N2 - Autophagy, that is directly triggered by invaded pathogens and indirectly triggered by IFN-γ, acts as a defense by mediating intracellular microbial recognition and clearance. In addition, autophagy contributes to inflammation by facilitating an IFN-γ response and signal transduction. For immune escape, downregulated autophagy may be a strategy used by microbes.

AB - Autophagy, that is directly triggered by invaded pathogens and indirectly triggered by IFN-γ, acts as a defense by mediating intracellular microbial recognition and clearance. In addition, autophagy contributes to inflammation by facilitating an IFN-γ response and signal transduction. For immune escape, downregulated autophagy may be a strategy used by microbes.

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UR - http://www.scopus.com/inward/citedby.url?scp=80052363444&partnerID=8YFLogxK

U2 - 10.1016/j.micinf.2011.05.008

DO - 10.1016/j.micinf.2011.05.008

M3 - Short survey

C2 - 21664983

AN - SCOPUS:80052363444

SN - 1286-4579

VL - 13

SP - 888

EP - 894

JO - Microbes and Infection

JF - Microbes and Infection

IS - 11

ER -

Autophagy facilitates an IFN-γ response and signal transduction

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All Science Journal Classification (ASJC) codes

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